Additionally, the anti-tumour effects are performed by: (i) glucose consumption decrease; (ii) production of ATP and lactic acid decrease; (iii) kinases’ activities, such as phosphofructokinase (PFK), hexokinase (HK), and pyruvate kinase (PK) inhibition; and (iv) expression of glucose cotransporter-1 (GLUT1), monocarboxylate transporter 1 (MCT-1), and MCT4 inhibition [131]. This evidence concerns the gene SLC16A1 and neoplasm.