AKT1 and glioblastoma: This study also demonstrated that miR-203 potentially controls DNA damage repair via the PI3K/AKT (Phosphoinositide 3-kinases/AKT) and JAK/STAT3 (Janus Kinase 2/Signal transducer and activator of transcription 3) pathways and may collectively contribute to modulation of radiosensitivity in glioblastoma cells by inhibiting the repair of DNA damage and EMT [38].