GNAS-AS1 and breast carcinoma: Ectopic miR-296-5p expression reduced telomerase activity, drove telomere shortening, and caused proliferation defects by enhancing senescence and apoptosis in breast cancer cell lines, but the expression of Nespas and miR-296-5p was reduced in human basal-type breast cancer tissue, contributing to the higher aggressiveness of this type of the breast cancer.