Incubation of HepG2 cells with dapagliflozin, canagliflozin and empagliflozin had no effect on cancer cell survival [81,86] and adhesion capacity [81], while the cell sensitivity to dapagliflozin was induced after UDP Glucuronosyltransferase Family 1 Member A9 (UGT1A9) silencing, indicated by an increased number of floating HepG2 cells; of note, UGT1A9 metabolizes and deactivates dapagliflozin [81]. Here, UGT1A9 is linked to cancer.