miRNA-17 is mainly linked to the differentiation and activation of Th1, an essential cell in autoimmune disease, promoting the production of interferon-γ [56] that signals through the JAK/STAT pathway; its inhibition is an effective therapy for various autoimmune disorders, and the JAK inhibitor has been used to reverse AA in humans and mice [11]. Here, SOAT1 is linked to Autoimmunity.