TTR also possesses intrinsic amyloidogenic features and its aggregation is linked to several amyloid disorders including senile systemic amyloidosis (SSA), familial amyloid cardiomyopathy (FAC), and familial amyloid polyneuropathy (FAP)—a hereditary disease variant caused by one of over 100 known point mutations in the ttr gene [58,59,60]. This evidence concerns the gene TTR and familial amyloid neuropathy.