By contrast, in T-cell lymphoma (CTCL), Günther et al. found that CCL18, which is derived from CD209+ macrophages in mycosis fungoides (MF), the most frequent form of cutaneous T-cell lymphoma (CTCL), did not induce proliferation in CTCL cell lines, including Hut78, SeAx, and MyLa; rather, CCL18 was found to inhibit the proliferation of both SeAx and MyLa [80]. This evidence concerns the gene CCL18 and mycosis fungoides.