For instance, Nie et al. showed that CCL5 derived from breast phyllode tumors was involved in TAM recruitment by binding with CCR5 on the membranes of TAMs, and the CCL5 inhibitor, maraviroc, was shown to attenuate TAM recruitment and suppress malignant progression [15], indicating that targeting CCR5 in TAMs might represent a potential strategy for decreasing recruitment in malignant breast phyllode tumors. The gene discussed is CCL5; the disease is breast phyllodes tumor.