-Inhibited the recombinant DNMT1-Reactivated the expression of a DNA methylation-silenced gene, E-cadherin, in MDA-MB-231 cells-Suppressed the proliferation of MCF-7 breast and LNCaP prostate cancer cells through apoptosis induction-Inhibited DNMT activity by competing with cytosine binding⇒ Kazinol Q has a remarkable capacity to cause ROS, which, when combined with DNMT1 inhibition, could explain the role of kazinol Q on cancer cell proliferation and apoptosis. The gene discussed is CDH1; the disease is prostate cancer.