Inhibited the cell growth in a dose- and time-dependent manner in both breast cancer cell linesAttenuated the invasive capacity and anchorage independent growth by 7 μmActivated the tumor suppressor gene SEMA3A epigenetically in breast cancer cellsDecreased the DNMT3A occupancy within SEMA3A promoterIncreased the nuclear factor 1C occupancy at SEMA3A promoterIncreased the SALL3 expression by 1.5-fold upon 9-day treatment of MCF10CA1a cells with 7 μm PTS. This evidence concerns the gene SALL3 and breast carcinoma.