Inhibited the cell growth in a dose- and time dependent manner in both cell linesIncreased the methylation at CpG#2 and/or CpG#1 in both cancer cellsDownregulated the MAML2 in both cancer cell linesDecreased the expression of the tested genes in MCF10CA1h cellsAltered DNA methylation patterns in breast cancer cellsReduced invasive properties of breast cancer cells and their ability to anchorage-independent growth. Here, MAML2 is linked to cancer.