The upregulation of mitochondrial enzymes, such as ATP synthase F1 subunit alpha (Atp5a1), ATP synthase F1 subunit gamma (Atp5c1), and ATP synthase peripheral stalk-membrane subunit b (Atp5f1), in AIP mice indicated that these organelles played a critical role during acute manifestations [57]. Here, ATP5PB is linked to autoimmune pancreatitis.