NKG2C+CD57+ cell proliferation was distinctly dependent on ITP platelet presence in both FcεRIγ− and FcεRIγ+ pools; of note, and limitedly to the FcεRIγ− component, the more immature NKG2C+CD57− subset showed a more marked expansion in the presence of ITP platelets (Figure 5B upper and lower panel, respectively). This evidence concerns the gene KLRC2 and autoimmune thrombocytopenic purpura.