FCGR3A and autoimmune thrombocytopenic purpura: The FcεRIγ−NKG2C+CD57+ memory subset was further amplified in HCMV-seropositive patients affected by ITP, despite that anti-HCMV IgG levels were comparable between patients and healthy controls (Capuano C. et al., unpublished data), suggesting for the first time the possibility that opsonizing auto-antibodies can also effectively influence the size of this CD16-hyperresponsive memory subset in humans.