In addition, regression of NAFLD has been shown to normalize the production of adipokines (adiponectin, omentin) that suppress vascular inflammation, endothelial adhesion of monocytes, expression of adhesion modules, inhibit the development of atherogenic dyslipidemia [49], and also lead to a decrease in the intima-media thickness [49]. Here, ADIPOQ is linked to metabolic dysfunction-associated steatotic liver disease.