In KRAS-mutated, but not in KRAS-wild type lung cancer cells, AURKA depletion selectively blocks cell proliferation and expression of mitotic regulators (AURKA, CCNB1, HURP, TACC3, and PLK1), though AURKA overexpression is insufficient to rescue all the effects of FOSL1-knockdown in KRAS-mutated cells [37]. This evidence concerns the gene FOSL1 and lung cancer.