FOSL1 and breast neoplasm: Downregulation of miR-19a-3p participates in Fra-1 accumulation in TAMs (Tumor-Associated Macrophages) recruited to breast tumors microenvironment, in which the miR-19a-3p-Fra-1-Stat3 pathway controls the macrophage polarization towards the pro-neoplastic immunosuppressive M2 phenotype [18].