DGAT1 and steatosis: Mechanisms allowing this steatosis involve the increased expression of enzymes implicated in TG synthesis, such as DGAT1 (diacylglycerol O-acyltransferase 1) and lipin-1, and of genes involved in lipid droplets formation (Cidea, Cidec, Plin2, -3, and -5, and Hilpa), concomitant to a reduction in VLDL-TG secretion.