This improvement was accompanied by a decrease in malondialdehyde (MDA) levels, a reverse in the depletion of glutathione (GSH) levels observed in mice with intestinal ischemia and reperfusion, inhibition of pro-inflammatory factors expressions, such as TNF-α and IL-1β, and a decrease in myeloperoxidase (MPO) activity in the intestinal tissue [222]. This evidence concerns the gene MPO and ischemia.