The objective of this study was instead to define the mechanistic interaction of two drugs: Fdb, which stimulates the cells via adaptive autophagy, and IFN-α, which induces regulation of STAT1 signaling in certain cell types that are at a high risk of induced death during chemotherapy that kills the cancer cells or during antiviral drug treatment that kills infected cells as well as normal cells. Here, STAT1 is linked to cancer.