The mRNA expression of the angiogenic factors, eNOS, and ANG2, and superoxide dismutase 2 (SOD2), mitochondrial antioxidant, were significantly decreased in GLA‐mutant kidney organoids, which is compatible with the phenotypes of vasculopathy in Fabry disease (Figure 7h).[47, 48]. This evidence concerns the gene SOD2 and Fabry disease.