TRPV1 and glioma: A hallmark study (Stock et al., 2012) showed that fatty acyl‐ethanolamides (i.e., AEA and molecular congeners) released from neural progenitor cells of the mouse brain can activate transient receptor potential vanilloid subfamily member‐1 (TRPV1) located on the ER of neighboring glioma cells, thereby inducing Ca2+ release into the cytoplasm (Figure 2).