It provided some associated key genes and pathways to understand the molecular mechanisms in IUGR-induced metabolic syndrome from GO and KEGG pathway analyses such as Angptl4, Ehhadh, Ppargc1a, Crat, Slc2a1, Fabps, Acsls, and so on, as well as the PPAR signaling pathway, which had an essential role in IUGR. This evidence concerns the gene ANGPTL4 and fetal growth restriction.