It provided some associated key genes and pathways to understand the molecular mechanisms in IUGR-induced metabolic syndrome from GO and KEGG pathway analyses such as Angptl4, Ehhadh, Ppargc1a, Crat, Slc2a1, Fabps, Acsls, and so on, as well as the PPAR signaling pathway, which had an essential role in IUGR. Here, PPARA is linked to fetal growth restriction.