In this study, the analysis of GO MF revealed that DEGs mainly enriched in organic anion transmembrane transporter activity such as upregulated Ctns, Slc2a1, Slc25a32, Slc7a11, Slc16a10, Slc38a2, Slc7a2, Slc25a15, Slc7a1, and Slc25a29 and downregulated Slc13a4, Slc17a2, Slc17a4, Slc26a1, Slc16a7, Slc25a42, and Slc25a23. The findings indicated that the change of transmembrane transporter-related regulatory genes might be an important event in the metabolic syndrome induced by IUGR. The gene discussed is CTNS; the disease is fetal growth restriction.