CD8A and myocarditis: Here, a new glance was lanced at these data,43 focusing on a group of molecules that may contribute to inflammation-related alterations playing a role in major biological processes as cell migration (CCL3, CCL5, CXCL10, CXCL11, CXCR3), myocarditis formation (CD3ε, CD8a, CD4, CD19), inflammation (IFNγ, IL-10, IL-12a, IL-15, IL2R, IL-1Ra, IL-6, IL-7), and cytotoxic activity of T-cells (CTL; granzyme, perforin1).