This specific mechanism could probably be more frequent in some specific genetic etiologies of DCM, such as pathogenic mutations in desmosomal genes, Desmin (DESM), Filamin C (FLNC), and Lamin (LMNA) genes, leading to an overlap between the DCM phenotype and specific forms of arrhythmogenic cardiomyopathy (AC) with biventricular involvement [14, 15]. This evidence concerns the gene LMNA and familial dilated cardiomyopathy.