With regard to basal-like breast cancer, which is a type of TNBC, Tell et al. [43] showed that STAT3 signaling is associated with the expression of multiple genes in basal-like, but not luminal A or B, breast cancer, and that targets of STAT3 signaling in basal-like breast cancer are largely immunological and inflammatory mediators. This evidence concerns the gene STAT3 and breast carcinoma.