SOS2 and non-small cell lung carcinoma: A recent study found that miR-148a-3p, which is a tumor suppressor, is often downregulated in NSCLC cells and can inhibit the proliferation and epithelial-mesenchymal transition processes of NSCLC by regulating SOS2/MAPK/ERK signal transduction, thereby providing new insight into the pathogenesis of NSCLC [68].