For instance, via activating phosphatidylinositol 3‐kinase/protein kinase B (PI3K/AKT) and mitogen‐activated protein kinases/extracellular signal‐regulated kinase (MAPK/ERK) signaling pathways, KIF2A can facilitate lung adenocarcinoma cell proliferation and suppress apoptosis13; by means of suppressing microRNA (miR)‐206, KIF2A promotes ovarian cancer cell proliferation and migration14; meanwhile, KIF2A also contributes to osteosarcoma cell (MG‐63 and U2OS cells) proliferation, migration and invasion.15 This evidence concerns the gene KIF2A and lung adenocarcinoma.