Due to the key role of BACH1 in different pathological scenarios such as chronic conditions and cancer, we tested whether CDDO-Me and CDDO-TFEA also reduced BACH1 levels in various relevant cell lines such as the immortalised human proximal tubular kidney cell line HK2, the human hepatic stellate cell line LX2 or the lung cancer cell lines H1299 and A549. The gene discussed is BACH1; the disease is lung carcinoma.