2018) and its knockout induces G1-phase cell cycle arrest in cancer cells (Zhao et al. 2020). Stat signaling is involved in numerous processes in the cell, including a centrosome doubling during cell cycle progression in CHO cells (Metge et al. 2004), and is obviously not required when the cells do not proliferate, which may explain the down-regulation of Stat protein in the “feed.” The gene discussed is SOAT1; the disease is cancer.