VIM and breast cancer: The mechanisms related to their therapeutic effects include inhibition of PI3K/Akt and PPTG1 signaling pathways, activation of LKB1-AMPK-P38MAPK-p53-survivin cascade resulting in cell death, and increased caspase-3-mediated vimentin hydrolysis leading to the death of breast cancer cells (164–167), indicating that it achieves the purpose of breast cancer treatment by regulating multiple signaling pathways.