Interestingly, under the inflammatory conditions induced by Poly(I:C), this thymic CXCL13 overexpression model gets a strong recruitment of B cells to the thymus, and is more susceptible to developing MG, as demonstrated by elevated clinical signs, anti-AChR antibody levels, and thymic GCs-like structures, when compared with wild-type mice (66). The gene discussed is CXCL13; the disease is myasthenia gravis.