However, recent findings demonstrated that ZIKV still showed effective invasion, infection, and replication in AXL-depleted cerebral organoids (Wells et al., 2016), neural progenitor cells (NPCs; Wells et al., 2016), and murine models (Wang et al., 2017b; Li et al., 2017c), suggesting that AXL may not serve as the exclusive receptor involved and that its dependency for viral entry may be cell-type or model specific. This evidence concerns the gene AXL and infection.