They indicated that MALAT1 by targeting miR-26a/26b promoted FUT4-associated fucosylation, stimulated the PI3K/AKT/mTOR pathway, and increased CRC cell proliferation and tumorigenesis (MALAT1/miR-26a/26b/FUT4 axis) [94]. The gene discussed is MALAT1; the disease is colorectal carcinoma.