A prominent aspect of CKD is altered mineral metabolism, where hyperphosphatemia and excess serum FGF23 are factors associated with inflammation, anemia, and mortality (Mehta et al., 2017; Munoz Mendoza et al., 2017; Navarro-González et al., 2009; Tran et al., 2016). This evidence concerns the gene FGF23 and chronic kidney disease.