Furthermore, phosphorylation levels of NF-κB, which is activated downstream of inflammasomes to promote gastric tumorigenesis in the gp130F/F model (29), were also similar between gp130F/F and gp130F/F:Nlrp3-/- gastric tumor or non-tumor samples (Figures 4A, D). This evidence concerns the gene NFKB1 and gastric neoplasm.