Moreover, the beneficial results obtained for the skeletal muscle function with K‐rhein and 78c, two CD38 inhibitors (Hogan et al, 2019) on two different models of DMD, namely the mdx and the mdx/utr−/− mice, and the effect of isatuximab on human DMD myotubes clearly reassert our results on CD38 deletion in mdx mice. Here, CD38 is linked to Duchenne muscular dystrophy.