A recent study showed that SMAD1 was highly expressed in RRMM cells and proved that SMAD1 mediated MM drug resistance by regulating of NF‐κB1/TNFAIP8 and ID1‐p21/p27 axes,61 strongly supporting the analysis result from our scRNA‐seq data. Here, NFKB1 is linked to Miyoshi myopathy.