reported lower placental expression of DLK1, primarily in the endothelium and cytotrophoblast, following immunohistochemistry of 40 placental biopsies of healthy pregnant women and speculated that aberrant cytotrophoblast development and endothelial dysfunction in preeclampsia may contribute to reduced placental expression of DLK1 in SGA fetuses. The gene discussed is DLK1; the disease is endothelial dysfunction.