In that aspect, the similarities of clinical manifestations and histopathological characteristic between FXTAS, NIID, OPDM, and OPML question whether CGG repeats also located in sequences predicted as non-coding, such as the LOC642361/NUTM2B-AS1 locus or the 5′UTR of the LRP12 and GIPC1 genes, are similarly translated into novel and potentially pathogenic polyG proteins. This evidence concerns the gene GIPC1 and neuronal intranuclear inclusion disease.