In that aspect, the similarities of clinical manifestations and histopathological characteristic between FXTAS, NIID, OPDM, and OPML question whether CGG repeats also located in sequences predicted as non-coding, such as the LOC642361/NUTM2B-AS1 locus or the 5′UTR of the LRP12 and GIPC1 genes, are similarly translated into novel and potentially pathogenic polyG proteins. Here, NUTM2B is linked to neuronal intranuclear inclusion disease.