Conversely, cMyBP-C peptides 302A and 302S peptides did improve maximal ATPase activity in myofibrils from infarcted hearts at week 1 post-MI from 60.9 ± 5.3 nmol Pi/min/mg (sham, n = 15) to 87.9 ± 7.1 nmol Pi/min/mg (302A, n = 12; p < 0.05) and 91.3 ± 6.2 nmol Pi/min/mg (302S, n = 12; p < 0.05), respectively (Fig. 4C). This evidence concerns the gene MYBPC3 and myocardial infarction.