Ahn et al. also reported higher levelsof fucosylation of A1AT in HCC compared to hepatitis B virus (HBV)-infectedpatients.56 While decreased protein levelsof A1AT in NAFLD compared to control healthy subjects have been reportedin the past,46 we found that A1AT proteinlevels were statistically significantly higher in NASH compared tocontrols. The gene discussed is SERPINA1; the disease is metabolic dysfunction-associated steatotic liver disease.