This topography is similar to the reported distribution of reticular pseudodrusen in nonexudative AMD (27,28), which segregate with genetic complement defects such as CFI p.G119R and are associated with overlying RPE–BM effacement and rod dysfunction—unfortunately, image grading data are currently unavailable to verify this in our population (29,30). This evidence concerns the gene CFI and age-related macular degeneration.