The durable tumor control induced by NKG2A blockade was abolished upon NK cell depletion,14 which suggests that NK cells also contribute to NKG2A blockade‐mediated anti‐tumor response and thus provides another explanation for the improved tumor control but no ulterior Tex cell response upon NKG2A blockade plus PD‐L1 blockade observed in the study. Here, KLRC1 is linked to neoplasm.