Given that CrkII only interacted with Abl in response to infection (Fig. 6a), we next infected the aforementioned transfected BMDMs with ∆U∆T P. aeruginosa strain and assessed the interaction between ExoT–GFP and ExoT(G−A−)–GFP with Crk and Abl in the aforementioned transfected BMDMs under infection. The gene discussed is ABL1; the disease is infection.