Expression analysis also determined cell identity genes whose enhancers, via rearrangements/translocations, were found to activate oncogenes such as MYCN, MYC, and TERT [44] through a mechanism of enhancer hijacking that juxtaposes ectopic enhancers or SEs controlled by a specific CRC to the gene transcriptional unit [80, 81]. Here, MYCN is linked to colorectal carcinoma.