TWIST1 and neuroblastoma: EMT was controlled by effectors such as SNAI1/2, ZEB1/2, TWIST1/2 leading to the expression of E-cadherin and claudins, while in NB NMT was promoted by PRRX1 or NOTCH3-IC overexpression and GATA3 ablation leading to the expression of mesenchymal markers such as vimentin and fibronectin [100].