Parts of the data have previously been used in organoid-studies of epithelial ISG15 [26] and SLC6A4 [14] regulation and signaling in IBD, demonstrating that the data presented in full here can act as a valuable tool for further studies on epithelial involvement in IBD pathophysiology and the contrast of epithelial UC and CD expression, while also confirming in vivo relevance of observations from organoid studies. The gene discussed is SLC6A4; the disease is inflammatory bowel disease.