Thick ascending limb‐specific disruption of cilia (using NKCC2‐Cre to achieve Ift88 KO) caused salt‐sensitive hypertension and enhanced tubuloglomerular feedback in male mice without cysts (females were not studied and renal Na+ transporters were not measured) (Song et al., 2017), suggesting that thick ascending limb cilia disruption, independent of cysts and at least in males, elicits hypertension. The gene discussed is IFT88; the disease is hypertensive disorder.