IKZF1 alterations have been shown to contribute to a stem‐like, glucocorticoid‐resistant, and adhesive phenotype with increased expression of, that is, integrin alpha 5 and L‐selectin in Ph+ BCP‐ALL [55], which is in line with our findings of upregulation of cell adhesion‐related genes in CD34‐positive leukemias and poor outcome of the CD34‐positive, Ph‐like patients. The gene discussed is SELL; the disease is acute lymphoblastic leukemia.