Their expression and downstream function mechanisms (target genes identified by ceRNA or co-expression analysis) still needed further confirmation by more high-throughput datasets and wet experiments (including PCR, Western blot, overexpression or silencing of genes, dual-luciferase reporter assay, proliferation, apoptosis, invasion and metastasis analyses) in in vitro (different cell lines) and in vivo lung cancer models induced by different types of CNTs, especially for those first reported in our study, such as RPL23AP64, ZNF826P and TMEM198B. This evidence concerns the gene TMEM198B and lung carcinoma.