To analyze a potential mechanism behind the altered phenotype of endometriotic cells due to Musashi double knockdown, the RT-qPCR of genes involved in Notch signaling (Notch-1, Notch-2, Notch-3, HES-1, HES-2, HEY-1, and HEY-2), in maintaining stem cell functions (KLF-4, OCT-4, and SOX-2) and in the pathogenesis of endometriosis (LIFR, Tert, IFITM1, and FOXA2) [1] was analyzed (Figure 6). The gene discussed is HES2; the disease is endometriosis.