Wroblewski et al. [51] reported that, compared to organoids infected with a CagA-negative mutant, murine gastric organoids infected with CagA-positive H. pylori 7.13 strain showed redistribution of tight junction proteins such as β-catenin and claudin 7—a tight junction protein that has been previously associated with other types of cancers [51]. The gene discussed is S100A8; the disease is cancer.