Such event was indeed supported by the higher levels of myogenin and lower levels of PAX7, found in BMD2 patients; and was further corroborated by the disease and biofunction analysis, provided by IPA, utilizing the four datasets described above, indicating that the survival of stem cell lines is increased in BMD2 compared to BMD1, and is inhibited in DMD. This evidence concerns the gene MYOG and Duchenne muscular dystrophy.