TPP1 and pulmonary fibrosis: The stress-induced pulmonary senescence and fibrotic onset are targetable through animal inhalation of either recombinant cDNA coding for the telomere protein TPP1 or a small 8-mer peptide (telomere dysfunction inhibitor or TELODIN) to prevent telomere uncapping, indicating an early effective intervention strategy on the mechanisms of telomere dysfunction and subsequent cellular senescence prior to differentiation disorder of pulmonary fibrosis [35,213].