In addition to the anti-TGF-β activity of PM014, it also acts via the inhibition of the expression of cytokines (IL-6, IL-13, IL-1b, and TGF-β), chemokines (MIP1a, MCP1, and CCL4), and fibrosis-related genes (Col3al and Fn1) in radiation-induced PF mice [235]. This evidence concerns the gene IL1B and pemphigus foliaceus.